The simultaneous loss of Arx and Pax4 genes promotes a somatostatin-producing 
cell fate specification at the expense of the alpha- and beta-cell lineages in 
the mouse endocrine pancreas

Collombat, P.; Hecksher-Soerensen, J.; Broccoli, V.; Krull, J.; Ponte, I.; Mundiger, T.; Smith, J.; Gruss, P.; Serup, P.; Mansouri, A.

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The simultaneous loss of Arx and Pax4 genes promotes a somatostatin-producing cell fate specification at the expense of the alpha- and beta-cell lineages in the mouse endocrine pancreas

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Collombat, P.
Research Group of Molecular Cell Differentiation, MPI for biophysical chemistry, Max Planck Society;

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Krull, J.
Research Group of Molecular Cell Differentiation, MPI for biophysical chemistry, Max Planck Society;

Gruss, P.
Max Planck Society;

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Mansouri, A.
Research Group of Molecular Cell Differentiation, MPI for biophysical chemistry, Max Planck Society;

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Collombat, P., Hecksher-Soerensen, J., Broccoli, V., Krull, J., Ponte, I., Mundiger, T., et al. (2005). The simultaneous loss of Arx and Pax4 genes promotes a somatostatin-producing cell fate specification at the expense of the alpha- and beta-cell lineages in the mouse endocrine pancreas. Development, 132(13), 2969-2980. Retrieved from http://dev.biologists.org/cgi/content/full/132/13/2969.

Cite as: https://hdl.handle.net/11858/00-001M-0000-0012-E8A3-6

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