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Hypertonicity-induced cation channels (HICCs) in HepG2 cells: architecture and role in proliferation vs. apoptosis

MPS-Authors
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Koos,  Björn
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Christmann,  Jens
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Plettenberg,  Sandra
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Käding,  Domenic
Chemical Genomics Center of the MPS, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Becker,  Julia
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Keteku,  Melody
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Klein,  Christian
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Imtiaz,  Sarah
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Janning,  Petra
Abt. IV: Chemische Biologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Bastiaens,  Philippe I. H.
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Wehner,  Frank
Abt. II: Systemische Zellbiologie, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Citation

Koos, B., Christmann, J., Plettenberg, S., Käding, D., Becker, J., Keteku, M., et al. (2018). Hypertonicity-induced cation channels (HICCs) in HepG2 cells: architecture and role in proliferation vs. apoptosis. The Journal of Physiology - London, 1-35. doi:10.1113/JP275827.


Cite as: https://hdl.handle.net/21.11116/0000-0000-62DA-C
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