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The target of rapamycin kinase affects biomass accumulation and cell cycle progression by altering carbon/nitrogen balance in synchronized Chlamydomonas reinhardtii cells

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Jueppner,  J.
Experimental Systems Biology, Department Willmitzer, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Mubeen,  U.
Experimental Systems Biology, Department Willmitzer, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Leisse,  A.
Experimental Systems Biology, Department Willmitzer, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Caldana,  C.
Metabolic Regulation of Plant Growth, Department Willmitzer, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Wiszniewski,  A.A.G.
Experimental Systems Biology, Department Willmitzer, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Steinhauser,  D.
Systems Metabolomics, Department Willmitzer, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Giavalisco,  P.
Experimental Systems Biology, Department Willmitzer, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Citation

Jueppner, J., Mubeen, U., Leisse, A., Caldana, C., Wiszniewski, A., Steinhauser, D., et al. (2018). The target of rapamycin kinase affects biomass accumulation and cell cycle progression by altering carbon/nitrogen balance in synchronized Chlamydomonas reinhardtii cells. The Plant Journal, 93(2), 355-376. doi:10.1111/tpj.13787.


Cite as: https://hdl.handle.net/21.11116/0000-0000-3130-2
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