Neuronal Deletion of Kmt2a/Mll l Histone Methyltransferase in Ventral Striatum 
is Associated with Defective Spike-Timing-Dependent Striatal Synaptic 
Plasticity, Altered Response to Dopaminergic Drugs, and Increased Anxiety

Shen, Erica Y.; Jiang, Yan; Javidfar, Behnam; Kassim, Bibi; Loh, Yong-Hwee E.; Ma, Qi; Mitchell, Amanda C.; Pothula, Venu; Stewart, A. Francis; Ernst, Patricia; Yao, Wei-Dong; Martin, Gilles; Shen, Li; Jakovcevski, Mira; Akbarian, Schahram

doi:10.1038/npp.2016.144

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Neuronal Deletion of Kmt2a/Mll l Histone Methyltransferase in Ventral Striatum is Associated with Defective Spike-Timing-Dependent Striatal Synaptic Plasticity, Altered Response to Dopaminergic Drugs, and Increased Anxiety

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Jakovcevski, Mira
Dept. Stress Neurobiology and Neurogenetics, Max Planck Institute of Psychiatry, Max Planck Society;

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Shen, E. Y., Jiang, Y., Javidfar, B., Kassim, B., Loh, Y.-H.-E., Ma, Q., et al. (2016). Neuronal Deletion of Kmt2a/Mll l Histone Methyltransferase in Ventral Striatum is Associated with Defective Spike-Timing-Dependent Striatal Synaptic Plasticity, Altered Response to Dopaminergic Drugs, and Increased Anxiety. NEUROPSYCHOPHARMACOLOGY, 41(13), 3103-3113. doi:10.1038/npp.2016.144.

Cite as: https://hdl.handle.net/11858/00-001M-0000-002C-9C41-E

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