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Journal Article

Regulation of back−propagating action potentials in hippocampal neurons

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Hoffman, Dax A.
Department of Cell Physiology, Max Planck Institute for Medical Research, Max Planck Society;
Synaptic Plasticity and Behaviour, Max Planck Institute for Medical Research, Max Planck Society;

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Citation

Johnston, D., Hoffman, D. A., Colbert, C. M., & Magee, J. C. (1999). Regulation of back−propagating action potentials in hippocampal neurons. Current Opinion in Neurobiology, 9(3): 231549, pp. 288-292. doi:http://dx.doi.org/10.1016/S0959-4388(99)80042-7.

Abstract

Protein kinase C has recently been shown to modulate the slow recovery from inactivation of Na+ channels in apical dendrites of hippocampal CA1 pyramidal neurons. Moreover, dendritic, A−type K+ channels have been found to be modulated by protein kinases A and C and by mitogen−activated protein kinase. The electrical signalling ability of these dendrites is thus highly regulated by a number of neurotransmitters and second−messenger systems