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The paramutated SULFUREA locus of tomato is involved in auxin biosynthesis

MPG-Autoren
http://pubman.mpdl.mpg.de/cone/persons/resource/persons97392

Schöttler,  M. A.
Photosynthesis Research, Department Bock, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons97077

Bock,  R.
Organelle Biology and Biotechnology, Department Bock, Max Planck Institute of Molecular Plant Physiology, Max Planck Society;

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Volltexte (frei zugänglich)

Ehlert-2008-The paramutated SULF.pdf
(beliebiger Volltext), 2MB

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Zitation

Ehlert, B., Schöttler, M. A., Tischendorf, G., Ludwig-Mueller, J., & Bock, R. (2008). The paramutated SULFUREA locus of tomato is involved in auxin biosynthesis. Journal of Experimental Botany, 59(13), 3635-3647. doi:10.1093/Jxb/Ern213.


Zitierlink: http://hdl.handle.net/11858/00-001M-0000-0014-27C4-0
Zusammenfassung
The tomato (Solanum lycopersicum) sulfurea mutation displays trans-inactivation of wild-type alleles in heterozygous plants, a phenomenon referred to as paramutation. Homozygous mutant plants and paramutated leaf tissue of heterozygous plants show a pigment-deficient phenotype. The molecular basis of this phenotype and the function of the SULFUREA gene (SULF) are unknown. Here, a comprehensive physiological analysis of the sulfurea mutant is reported which suggests a molecular function for the SULFUREA locus. It is found that the sulf mutant is auxin-deficient and that the pigment-deficient phenotype is likely to represent only a secondary consequence of the auxin deficiency. This is most strongly supported by the isolation of a suppressor mutant which shows an auxin overaccumulation phenotype and contains elevated levels of indole-3-acetic acid (IAA). Several lines of evidence point to a role of the SULF gene in tryptophan-independent auxin biosynthesis, a pathway whose biochemistry and enzymology is still completely unknown. Thus, the sulfurea mutant may provide a promising entry point into elucidating the tryptophan-independent pathway of IAA synthesis.