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Journal Article

Impaired Binding of 14-3-3 to C-Raf in Noonan Syndrome Suggests New Approaches in Diseases with Increased Ras Signaling

MPS-Authors

Molzan,  Manuela
Max Planck Institute of Molecular Physiology, Max Planck Society;

Schumacher,  Benjamin
Max Planck Institute of Molecular Physiology, Max Planck Society;

Ottmann,  Corinna
Max Planck Institute of Molecular Physiology, Max Planck Society;

Weyand,  Michael
Max Planck Institute of Molecular Physiology, Max Planck Society;

Thiel,  Philipp
Max Planck Institute of Molecular Physiology, Max Planck Society;

Rose,  Rolf
Max Planck Institute of Molecular Physiology, Max Planck Society;

Rose,  Micheline
Max Planck Institute of Molecular Physiology, Max Planck Society;

Kuhenne,  Philipp
Max Planck Institute of Molecular Physiology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons98702

Kaiser,  Markus
Chemical Genomics Center of the MPS, Max Planck Institute of Molecular Physiology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons98707

Kuhlmann,  Jürgen
Sonstige Wissenschaftliche Organisationseinheiten, Max Planck Institute of Molecular Physiology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons98717

Ottmann,  Christian
Chemical Genomics Center of the MPS, Max Planck Institute of Molecular Physiology, Max Planck Society;

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Citation

Molzan, M., Schumacher, B., Ottmann, C., Baljuls, A., Polzien, L., Weyand, M., et al. (2010). Impaired Binding of 14-3-3 to C-Raf in Noonan Syndrome Suggests New Approaches in Diseases with Increased Ras Signaling. Molecular and Cellular Biology, 30(19): 1, pp. 4698-4711. Retrieved from http://dx.doi.org/10.1128/MCB.01636-09.


Cite as: http://hdl.handle.net/11858/00-001M-0000-0014-0140-4
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