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Correction of the iron overload defect in beta-2-microglobulin knockout mice by lactoferrin abolishes their increased susceptibility to tuberculosis

MPS-Authors
http://pubman.mpdl.mpg.de/cone/persons/resource/persons82134

Schaible,  Ulrich E.
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons81841

Collins,  Helen L.
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons81969

Kaufmann,  Stefan H. E.
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Citation

Schaible, U. E., Collins, H. L., Priem, F., & Kaufmann, S. H. E. (2002). Correction of the iron overload defect in beta-2-microglobulin knockout mice by lactoferrin abolishes their increased susceptibility to tuberculosis. Journal of Experimental Medicine, 196(11), 1507-1513.


Cite as: http://hdl.handle.net/11858/00-001M-0000-000E-C661-7
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