Increased resistance of LFA-1-deficient mice to lipopolysaccharide-induced 
shock/liver injury in the presence of TNF-alpha and IL-12 is mediated by IL-10: 
A novel role for LFA-1 in the regulation of the proinflammatory and 
anti-inflammatory cytokine balance

Emoto, Masashi; Emoto, Yoshiko; Brinkmann, Volker; Miyamoto, Mamiko; Yoshizawa, Izumi; Stäber, Manuela; van Rooijen, Nico; Hamann, Alf; Kaufmann, Stefan H. E.

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Increased resistance of LFA-1-deficient mice to lipopolysaccharide-induced shock/liver injury in the presence of TNF-alpha and IL-12 is mediated by IL-10: A novel role for LFA-1 in the regulation of the proinflammatory and anti-inflammatory cytokine balance

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Emoto, Masashi
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Emoto, Yoshiko
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Brinkmann, Volker
Core Facilities / Microscopy, Max Planck Institute for Infection Biology, Max Planck Society;

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Miyamoto, Mamiko
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Yoshizawa, Izumi
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Stäber, Manuela
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Kaufmann, Stefan H. E.
Department of Immunology, Max Planck Institute for Infection Biology, Max Planck Society;

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Citation

Emoto, M., Emoto, Y., Brinkmann, V., Miyamoto, M., Yoshizawa, I., Stäber, M., et al. (2003). Increased resistance of LFA-1-deficient mice to lipopolysaccharide-induced shock/liver injury in the presence of TNF-alpha and IL-12 is mediated by IL-10: A novel role for LFA-1 in the regulation of the proinflammatory and anti-inflammatory cytokine balance. Journal of Immunology, 171(2), 584-593.

Cite as: https://hdl.handle.net/11858/00-001M-0000-000E-C607-6

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