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Helicobacter pylori induces AGS cell motility and elongation via independent signaling pathways

MPS-Authors
http://pubman.mpdl.mpg.de/cone/persons/resource/persons82056

Moese,  Stefan
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons82156

Selbach,  Matthias
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons81827

Brinkmann,  Volker
Core Facilities / Microscopy, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons82047

Meyer,  Thomas F.
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons81796

Backert,  Steffen
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Citation

Moese, S., Selbach, M., Kwok, T., Brinkmann, V., Konig, W., Meyer, T. F., et al. (2004). Helicobacter pylori induces AGS cell motility and elongation via independent signaling pathways. Infection and Immunity, 72(6), 3646-3649.


Cite as: http://hdl.handle.net/11858/00-001M-0000-000E-C546-F
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