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The Helicobacter pylori CagA protein disrupts matrix adhesion of gastric epithelial cells by dephosphorylation of vinculin

MPS-Authors
http://pubman.mpdl.mpg.de/cone/persons/resource/persons82056

Moese,  Stefan
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons82156

Selbach,  Matthias
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons81827

Brinkmann,  Volker
Core Facilities / Microscopy, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons81968

Karlas,  Alexander
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons81796

Backert,  Steffen
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons82047

Meyer,  Thomas F.
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Citation

Moese, S., Selbach, M., Brinkmann, V., Karlas, A., Haimovich, B., Backert, S., et al. (2007). The Helicobacter pylori CagA protein disrupts matrix adhesion of gastric epithelial cells by dephosphorylation of vinculin. Cellular Microbiology, 9(5), 1148-1161. doi:10.1111/j.1462-5822.2006.00856.x.


Cite as: http://hdl.handle.net/11858/00-001M-0000-000E-C2B2-4
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