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The Helicobacter pylori CagA protein disrupts matrix adhesion of gastric epithelial cells by dephosphorylation of vinculin

MPS-Authors
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Moese,  Stefan
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Selbach,  Matthias
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Brinkmann,  Volker
Core Facilities / Microscopy, Max Planck Institute for Infection Biology, Max Planck Society;

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Karlas,  Alexander
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Backert,  Steffen
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Meyer,  Thomas F.
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Citation

Moese, S., Selbach, M., Brinkmann, V., Karlas, A., Haimovich, B., Backert, S., et al. (2007). The Helicobacter pylori CagA protein disrupts matrix adhesion of gastric epithelial cells by dephosphorylation of vinculin. Cellular Microbiology, 9(5), 1148-1161. doi:10.1111/j.1462-5822.2006.00856.x.


Cite as: https://hdl.handle.net/11858/00-001M-0000-000E-C2B2-4
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