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Helicobacter pylori-induced modification of the histone H3 phosphorylation status in gastric epithelial cells reflects its impact on cell cycle regulation

MPS-Authors
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Fehri,  Lina Fassi
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Rechner,  Cindy
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Janssen,  Sabrina
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Mak,  Tim Nam
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Holland,  Carsten
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Bartfeld,  Sina
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Brüggemann,  Holger
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Meyer,  Thomas F.
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Citation

Fehri, L. F., Rechner, C., Janssen, S., Mak, T. N., Holland, C., Bartfeld, S., et al. (2009). Helicobacter pylori-induced modification of the histone H3 phosphorylation status in gastric epithelial cells reflects its impact on cell cycle regulation. Epigenetics, 4(8), 577-586.


Cite as: https://hdl.handle.net/11858/00-001M-0000-000E-C06B-7
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