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Autophagy-independent function of MAP-LC3 during intracellular propagation of Chlamydia trachomatis

MPS-Authors
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Al-Younes,  Hesham M.
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Al-Zeer,  Munir A.
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Khalil,  Hany
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Gussmann,  Joscha
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Karlas,  Alexander
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Machuy,  Nikolaus
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Brinkmann,  Volker
Core Facilities / Microscopy, Max Planck Institute for Infection Biology, Max Planck Society;

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Braun,  Peter R.
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Meyer,  Thomas F.
Department of Molecular Biology, Max Planck Institute for Infection Biology, Max Planck Society;

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Citation

Al-Younes, H. M., Al-Zeer, M. A., Khalil, H., Gussmann, J., Karlas, A., Machuy, N., et al. (2011). Autophagy-independent function of MAP-LC3 during intracellular propagation of Chlamydia trachomatis. Autophagy, 7(8), 814-828. doi:10.4161/auto.7.8.15597.


Cite as: https://hdl.handle.net/11858/00-001M-0000-000E-BEBD-F
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