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Journal Article

Absence epilepsy, ataxia and locomotor deficits in mice lacking the pacemaker channel HCN2

MPS-Authors

Ludwig,  A
Max Planck Institute of Psychiatry, Max Planck Society;

Budde,  T
Max Planck Institute of Psychiatry, Max Planck Society;

Munsch,  T
Max Planck Institute of Psychiatry, Max Planck Society;

Pape,  HC
Max Planck Institute of Psychiatry, Max Planck Society;

Langebartels,  A
Max Planck Institute of Psychiatry, Max Planck Society;

Lancel,  M
Max Planck Institute of Psychiatry, Max Planck Society;

Wotjak,  C
Max Planck Institute of Psychiatry, Max Planck Society;

Moosmang,  S
Max Planck Institute of Psychiatry, Max Planck Society;

Stieber,  J
Max Planck Institute of Psychiatry, Max Planck Society;

Kuhbandner,  S
Max Planck Institute of Psychiatry, Max Planck Society;

Feil,  R
Max Planck Institute of Psychiatry, Max Planck Society;

Biel,  M
Max Planck Institute of Psychiatry, Max Planck Society;

Hofmann,  F
Max Planck Institute of Psychiatry, Max Planck Society;

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Citation

Ludwig, A., Budde, T., Munsch, T., Pape, H., Langebartels, A., Lancel, M., et al. (2002). Absence epilepsy, ataxia and locomotor deficits in mice lacking the pacemaker channel HCN2. Naunyn-Schmiedebergs Archives of Pharmacology, 365(Suppl. 1), 303-303.


Cite as: http://hdl.handle.net/11858/00-001M-0000-000E-A235-2
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