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Prolactin but not ACTH increases during sodium lactate-induced panic attacks

MPG-Autoren

Otte,  C
Max Planck Institute of Psychiatry, Max Planck Society;

Kellner,  M
Max Planck Institute of Psychiatry, Max Planck Society;

Arlt,  J
Max Planck Institute of Psychiatry, Max Planck Society;

Jahn,  H
Max Planck Institute of Psychiatry, Max Planck Society;

Holsboer,  F
Max Planck Institute of Psychiatry, Max Planck Society;

Wiedemann,  K
Max Planck Institute of Psychiatry, Max Planck Society;

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Zitation

Otte, C., Kellner, M., Arlt, J., Jahn, H., Holsboer, F., & Wiedemann, K. (2002). Prolactin but not ACTH increases during sodium lactate-induced panic attacks. Psychiatry Research, 109(2), 201-205.


Zitierlink: http://hdl.handle.net/11858/00-001M-0000-000E-A233-6
Zusammenfassung
Paradoxically, the pituitary-adrenal axis is not activated during sodium lactate-induced panic. We measured the response of another stress-sensitive hormone, prolactin, to standard lactate and placebo infusion in a double-blind randomised design in eight patients with panic disorder and eight matched normal controls. Prolactin release was significantly elevated (P<0.05) in panickers compared with non-panickers, whereas ACTH secretion was not activated at all. This differential stress response needs further investigation. (C) 2002 Elsevier Science Ireland Ltd. All rights reserve