de.mpg.escidoc.pubman.appbase.FacesBean
English
 
Help Guide Disclaimer Contact us Login
  Advanced SearchBrowse

Item

ITEM ACTIONSEXPORT

Released

Journal Article

Prolactin but not ACTH increases during sodium lactate-induced panic attacks

MPS-Authors

Otte,  C
Max Planck Institute of Psychiatry, Max Planck Society;

Kellner,  M
Max Planck Institute of Psychiatry, Max Planck Society;

Arlt,  J
Max Planck Institute of Psychiatry, Max Planck Society;

Jahn,  H
Max Planck Institute of Psychiatry, Max Planck Society;

Holsboer,  F
Max Planck Institute of Psychiatry, Max Planck Society;

Wiedemann,  K
Max Planck Institute of Psychiatry, Max Planck Society;

Locator
There are no locators available
Fulltext (public)
There are no public fulltexts available
Supplementary Material (public)
There is no public supplementary material available
Citation

Otte, C., Kellner, M., Arlt, J., Jahn, H., Holsboer, F., & Wiedemann, K. (2002). Prolactin but not ACTH increases during sodium lactate-induced panic attacks. Psychiatry Research, 109(2), 201-205.


Cite as: http://hdl.handle.net/11858/00-001M-0000-000E-A233-6
Abstract
Paradoxically, the pituitary-adrenal axis is not activated during sodium lactate-induced panic. We measured the response of another stress-sensitive hormone, prolactin, to standard lactate and placebo infusion in a double-blind randomised design in eight patients with panic disorder and eight matched normal controls. Prolactin release was significantly elevated (P<0.05) in panickers compared with non-panickers, whereas ACTH secretion was not activated at all. This differential stress response needs further investigation. (C) 2002 Elsevier Science Ireland Ltd. All rights reserve