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Journal Article

The high-affinity non-peptide CRH1 receptor antagonist R121919 attenuates stress-induced alterations in plasma oxytocin, prolactin, and testosterone secretion in rats

MPS-Authors
http://pubman.mpdl.mpg.de/cone/persons/resource/persons80390

Keck,  M. E.
Max Planck Institute of Psychiatry, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons80585

Welt,  T.
Max Planck Institute of Psychiatry, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons80449

Müller,  M. B.
AG Müller, Marianne, Florian Holsboer (Direktor), Max Planck Institute of Psychiatry, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons80418

Landgraf,  R.
AG Landgraf, Rainer, Florian Holsboer (Direktor), Max Planck Institute of Psychiatry, Max Planck Society;

http://pubman.mpdl.mpg.de/cone/persons/resource/persons80372

Holsboer,  F.
Florian Holsboer (Direktor), Max Planck Institute of Psychiatry, Max Planck Society;

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Citation

Keck, M. E., Welt, T., Müller, M. B., Landgraf, R., & Holsboer, F. (2003). The high-affinity non-peptide CRH1 receptor antagonist R121919 attenuates stress-induced alterations in plasma oxytocin, prolactin, and testosterone secretion in rats. Pharmacopsychiatry, 36(1), 27-31.


Cite as: http://hdl.handle.net/11858/00-001M-0000-000E-9F61-4
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