The high-affinity non-peptide CRH1 receptor antagonist R121919 attenuates 
stress-induced alterations in plasma oxytocin, prolactin, and testosterone 
secretion in rats

Keck, M. E.; Welt, T.; Müller, M. B.; Landgraf, R.; Holsboer, F.

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The high-affinity non-peptide CRH1 receptor antagonist R121919 attenuates stress-induced alterations in plasma oxytocin, prolactin, and testosterone secretion in rats

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Keck, M. E.
Max Planck Institute of Psychiatry, Max Planck Society;

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Welt, T.
Max Planck Institute of Psychiatry, Max Planck Society;

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Müller, M. B.
AG Müller, Marianne, Florian Holsboer (Direktor), Max Planck Institute of Psychiatry, Max Planck Society;

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Landgraf, R.
AG Landgraf, Rainer, Florian Holsboer (Direktor), Max Planck Institute of Psychiatry, Max Planck Society;

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Holsboer, F.
Florian Holsboer (Direktor), Max Planck Institute of Psychiatry, Max Planck Society;

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Keck, M. E., Welt, T., Müller, M. B., Landgraf, R., & Holsboer, F. (2003). The high-affinity non-peptide CRH1 receptor antagonist R121919 attenuates stress-induced alterations in plasma oxytocin, prolactin, and testosterone secretion in rats. Pharmacopsychiatry, 36(1), 27-31.

引用: https://hdl.handle.net/11858/00-001M-0000-000E-9F61-4

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