Alternative splicing of Rac1 generates Rac1b, a self-activating GTPase.

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Alternative splicing of Rac1 generates Rac1b, a self-activating GTPase.

Fiegen, D., Haeusler, L., Blumenstein, L., Herbrand, U., Dvorsky, R., Vetter, I. R., et al. (2004). Alternative splicing of Rac1 generates Rac1b, a self-activating GTPase. The Journal of Biological Chemistry, 279(6), 4743-4749. doi:10.1074/jbc.M310281200.

Item is Discarded

Basic (Discarded)

Date of Discard: 2018-08-16

Comment:

Creators:
Fiegen, Dennis
Haeusler, Lars−Christian
Blumenstein, Lars¹
Herbrand, Ulrike
Dvorsky, Radovan
Vetter, Ingrid R.
Ahmadian, Mohammad Reza

Affiliations:
1Department of Biomolecular Mechanisms, Max Planck Institute for Medical Research, Max Planck Society, ou_1497700

Dates: Submitted: 2003-09-16Accepted: 2003-11-18Published Online: 2003-11-18Date issued: 2004-02-06

Files: 1 File

Locators: 2 Locators

version ID: item_1996795_2

Item State: Discarded

Name of Context: Import Context of the MPI for Medical Research, Affiliated to: Max Planck Institute for Medical Research