ATM-dependent telomere loss in aging human diploid fibroblasts and DNA damage lead to the post-translational activation of p53 protein involving poly(ADP-ribose) polymerase.

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ATM-dependent telomere loss in aging human diploid fibroblasts and DNA damage lead to the post-translational activation of p53 protein involving poly(ADP-ribose) polymerase.

Vaziri, H., MD, RC, Davison, T., YS, CH, G., & Benchimol, S. (1997). ATM-dependent telomere loss in aging human diploid fibroblasts and DNA damage lead to the post-translational activation of p53 protein involving poly(ADP-ribose) polymerase. The European Molecular Biology Organization Journal, 16(19), 6018-6033. Retrieved from http://www.nature.com/cgi-taf/DynaPage.taf?file=/emboj/journal/v16/n19/abs/7590575a.htmldynoptions=doi1116847718.

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Date of Discard: 2020-03-16

Comment: dublette

Creators:
Vaziri, H
MD , RC , Davison, T¹
YS , CH ,, GG
Benchimol, S

Affiliations:
1Department Empirical Inference, Max Planck Institute for Biological Cybernetics, Max Planck Society, ou_1497795

Dates: Date issued: 1997

Files: 0 Files

Locators: 0 Locators

version ID: item_1794219_1

Item State: Discarded

Name of Context: Import Context of the MPI for Biological Cybernetics, Affiliated to: Max Planck Institute for Biological Cybernetics